Saturday, 11 July 2015

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NEUROFIBROMATOSIS

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NEUROFIBROMATOSIS

OUTLINE
INTRODUCTION
CASE SUMMARIES
CLASSIFICATION
CLINICAL FEATURES
MANAGEMENT
COMMENTARY
CASE PRESENTATION
CASE 1
NAME: BAB
AGE: 20yrs
OCCUPATION: STUDENT
TRIBE:KANURI
PC: Multiple Subcutaneous Swellings

HPC
10yr hx of progressive subcutaneous swelling, painless, non-itchy
Swelling on Nose & Lt upper arm, progressed more rapidly than others
No hx of Trauma, hearing or visual impairment, seizures.
No loss of function in any limb, no +ve family hx.
Not known hypertensive or diabetic.



O/E
A young man, not in any form of distress,
Multiple café au lait spots & axillary freckles
Multiple nodules with the larger ones on the Lt upper arm & the tip of the nose measuring 8x10cm & 6x8cm respectively
With a cord like consistency
No PLND
O/E contd
CNS No focal neurological deficit
Chest Clinically clear
ABD NAD
DIAGNOSIS Neurofibromatosis TypeI
INVESTIGATIONS
Tissue biopsy Consistent with neurofibroma
FBC & ESR, EUC, Urinalysis, Skull X-ray- All within normal limit.
TREATMENT
Pt had excision of the large nodules and rhinoplasty done.
CASE 2
                    BIODATA.
NAME-TZH
AGE-12yrs
OCCUPATION-STUDENT
RESIDENCE-NGURU
TRIBE-HAUSA
RELIGION-MUSLIM
HISTORY
PC- Swelling on the Lt eye X 3yrs
HPC- 3yr hx of painless progressive swelling on the Lt eye. No similar swelling on other part of the body
No hx of trauma to the eye
No associated visual or hearing impairment
No hx of seizures or functional loss in the limbs. Positive family hx(mother had similar nodules)
O/E
A young girl not in any distress with multiple café-au-lait spots.
Status Localis- Obvious swelling on the Lt eye measuring 3x4cm extending from upper eyelid to the angle of the eye. No attachment to skin or underlying structures
Tender pre-auricular nerve
No detectable loss in vision
O/E contd
CNS-No focal neurological deficit
CHEST- Clinically clear
ABD- NAD
DIAGNOSIS-Plexiform neurofibromatosis of the trigeminal nerve.
INVESTIGATIONS
Tissue biopsy- Neurogenic hamartoma consistent with neurofibromatosis.
FBC & ESR, EUC, Urinalysis, skull X-ray, within normal limit.
TREATMENT
Had excision biopsy
Did well post-op & was discharged home.
CLASSIFICATION
  Autosomal dominant disorder resulting from an abnormality in neural crest differentiation and migration
  2 major subtypes,namely;
  Type 1(peripheral neurofibromatosis)-commoner,incidence of 1:4000 live births,linked to a gene on chromosome 17
  Type 2(central neurofibromatosis)-rare,incidence of 1:50000 live births,linked to a gene on long arm of chromosome 22
DIAGNOSTIC CRITERIA for NEUROFIBROMATOSIS 1

2 or more of the following:
    6 or more café au lait macules>5mm in diameter in prepubertal indviduals,>15mm in post pubertal
    2 or more neurofibromas or 1 plexiform neurofibroma
    Axillary or inguinal freckling
    Optic glioma
    2 or more Lisch nodules

A distinctive osseous lesion such as sphenoid dysplasia,thining of long bone cortex, pseudoarthrosis,
First degree relative with neurofibromatosis 1


DIAGNOSTIC CRITERIA FOR NEUROFIBROMATOSIS-II
Any 1 of the following:
Bilateral masses of the 8th cranial nerve seen with appropriate imaging techniques
A first degree relative with NF-2 and either
§  Unilateral mass of the 8th cranial nerve or
§  2 of the following-neurofibroma, meningioma, glioma, schwannoma,juvenile cataracts

CLINICAL FEATURES

Neurocutaneos disorder affecting nervous system, skin, soft tissues and bones.
History
§  Cutaneous discolouratin occurring 1st 3yrs of life
§  Multiple skin nodules occurring at puberty
§  Physical symptoms-pain, pathological fractures, seizures, hearing or visual impairement, headaches.   
Physical examination

§  Café au lait spots
§  Neurofibromas- Cutaneous, Subcutaneous & Plexiform
                     - Soft or firm
                   - Brown, Pink or Skin coloured
§  Axillary/groin frecklings
§  Lisch nodules(iris hamartoma)
§  Bony involvement-
Pseudoarthrosis
Bowing of long bones
Kyphoscoliosis
Orbital defects e.g pulsating exopthalmos due to sphenoid dysplasia

§  Neurologic Abnormalities
Deafness
Blindness
Hydrocephalus
Increased ICP
Below average intelligence (25-40%)
Mental Retardation (5-10%)
§  Endocrine Disorders
Short stature & Growth abnormality
Sexual Precocity (3-5%)
Features of 2o hyperparathyroidism & phaeocromocytoma





DIFFERENTIALS:
§ McCune-Albright syndrome: polyosteotic fibrous dysplasia, precocious puberty, café au lait spots & endocrinopathies
§Multiple Endocrine Neoplasia
§Onchocerca Nodules



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